CDC admits death toll is inflated! Of 161,392 deaths ONLY 6% / 9,683 ARE DIRECTLY CAUSED BY COVID.

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9/22/20 update on C19 among students on campus since August, from 37 U.S. universities: Despite ~48,300 “+ C19 tests” near absence of reported C19 hospitalizations, and zero reported deaths (twitter.com)





Andrew Bostom

@andrewbostom
9/22/20 update on C19 among students on campus since August, from 37 U.S. universities: Despite ~48,300 “+ C19 tests” near absence of reported C19 hospitalizations, and zero reported deaths





3:00 PM · Sep 22, 2020

https://twitter.com/intent/like?ref...-among-studen/c/&tweet_id=1308496346454913026

 

[CorpsVet]

Lots of conflicting data, IMO

 

[_CY_]

Lots of conflicting data, IMO

please be more specific so I can track it down and make corrections if necessary
I'm pretty darn careful, each and every article is properly sourced and vetted for accuracy.

within this thread are some of the world's best C19 resources currently available anywhere.

 

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James Todaro, MD
@JamesTodaroMD
3/ While antibodies against COVID-19 may only last months, T cell immunity can remain protective for years. In a study of 23 people who survived SARS in 2003, every single one had memory T cells that recognized the SARS virus 17 years later. (Nature) https://nature.com/articles/s41586-020-2550-z…
12:19 PM · Aug 10, 2020

SARS-CoV-2-specific T cell immunity in cases of COVID-19 and SARS, and uninfected controls
https://www.nature.com/articles/s41586-020-2550-z

https://www.nature.com/articles/s41586-020-2550-z.pdf

Abstract
Memory T cells induced by previous pathogens can shape susceptibility to, and the clinical severity of, subsequent infections1. Little is known about the presence in humans of pre-existing memory T cells that have the potential to recognize severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Here we studied T cell responses against the structural (nucleocapsid (N) protein) and non-structural (NSP7 and NSP13 of ORF1) regions of SARS-CoV-2 in individuals convalescing from coronavirus disease 2019 (COVID-19) (n = 36). In all of these individuals, we found CD4 and CD8 T cells that recognized multiple regions of the N protein. Next, we showed that patients (n = 23) who recovered from SARS (the disease associated with SARS-CoV infection) possess long-lasting memory T cells that are reactive to the N protein of SARS-CoV 17 years after the outbreak of SARS in 2003; these T cells displayed robust cross-reactivity to the N protein of SARS-CoV-2. We also detected SARS-CoV-2-specific T cells in individuals with no history of SARS, COVID-19 or contact with individuals who had SARS and/or COVID-19 (n = 37). SARS-CoV-2-specific T cells in uninfected donors exhibited a different pattern of immunodominance, and frequently targeted NSP7 and NSP13 as well as the N protein. Epitope characterization of NSP7-specific T cells showed the recognition of protein fragments that are conserved among animal betacoronaviruses but have low homology to ‘common cold’ human-associated coronaviruses. Thus, infection with betacoronaviruses induces multi-specific and long-lasting T cell immunity against the structural N protein. Understanding how pre-existing N- and ORF1-specific T cells that are present in the general population affect the susceptibility to and pathogenesis of SARS-CoV-2 infection is important for the management of the current COVID-19 pandemic.

 

[_CY_]

This next part if true has HUGE implications.
https://www.redstate.com/michael_th...people-who-majored-in-journalism-at-breifing/

2003 SARS outbreak wasn’t much of an outbreak. It only infected around 8000 people worldwide (and killed less than 800). That comes to only around 0.0001% of the world’s population infected.

So if we only had crossover T-cell immunity to COVID-19 from SARS-CoV-1, while scientifically interesting, it wouldn’t have much practical import.

But it turns out that common cold coronaviruses that pretty much everyone has had can also provide T-cell immunity to COVID-19. In fact, the research shows that around half of us were likely immune to COVID-19 even before the virus that’s supposed to cause it ever existed!


[URL='https://twitter.com/JamesTodaroMD?ref_src=twsrc%5Etfw%7Ctwcamp%5Etweetembed%7Ctwterm%5E1292873241003003904%7Ctwgr%5Eshare_3&ref_url=https%3A%2F%2Fwww.redstate.com%2Fmichael_thau%2F2020%2F09%2F24%2Fwatch-trumps-new-covid-explain-covid-science-to-room-of-people-who-majored-in-journalism-at-breifing%2F']James Todaro, MD
@JamesTodaroMD[/URL]
Replying to @JamesTodaroMD
5/ In the same study, in 37 persons with no history of SARS or COVID-19 (negative serology and/or samples taken before COVID-19), over 50% had SARS-CoV-2 specific T cells. This is not surprising because there are at least 4 strains of coronaviruses that cause the "common cold".
12:19 PM · Aug 10, 2020

Presence of SARS-CoV-2 reactive T cells in COVID-19 patients and healthy donors
https://www.medrxiv.org/content/10.1101/2020.04.17.20061440v1

https://www.medrxiv.org/content/10.1101/2020.04.17.20061440v1.full.pdf

Abstract
The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has caused a rapidly unfolding pandemic, overwhelming health care systems worldwide1. Clinical manifestations of Corona-virus-disease 2019 (COVID-19) vary broadly, ranging from asymptomatic infection to acute respiratory failure and death2, yet the underlying physiological conditions and mechanisms for this high variability are still unknown. Also, the role of host immune responses in viral clearance and its involvement in pathogenesis remains unresolved. For SARS-CoV (2002/03), however, CD4+ T cell responses are generally associated with positive outcomes3,4, while cellular immune responses to SARS-CoV-2 have not yet been investigated. Here we describe an assay that allows direct detection and characterization of SARS-CoV-2 spike glycoprotein (S)-reactive CD4+ T cells in peripheral blood. We demonstrate the presence of S-reactive CD4+ T cells in 83% of COVID-19 patients, as well as in 34% of SARS-CoV-2 seronegative healthy donors, albeit at lower frequencies. Strikingly, in COVID-19 patients S-reactive CD4+ T cells equally targeted both N-terminal and C-terminal parts of S whereas in healthy donors S-reactive CD4+ T cells reacted almost exclusively to the Cterminal part that is a) characterized by higher homology to spike glycoprotein of human endemic "common cold" coronaviruses, and b) contains the S2 subunit of S with the cytoplasmic peptide (CP), the fusion peptide (FP), and the transmembrane domain (TM) but not the receptor-binding domain (RBD). S-reactive CD4+ T cells from COVID-19 patients were further distinct to those from healthy donors as they co-expressed higher levels of CD38 and HLA-DR, indicating their recent in vivo activation. Our study is the first to directly measure SARS-CoV-2-reactive T cell responses providing critical tools for large scale testing, in depth epitope mapping and characterization of potential cross-reactive cellular immunity to SARS-CoV-2. The presence of pre-existing SARS-CoV-2-reactive T cells in healthy donors is of high interest but larger scale prospective cohort studies are needed to assess whether their presence is a correlate of protection or pathology. Results of such studies will be key for a mechanistic understanding of the SARS-CoV-2 pandemic, adaptation of containment methods and to support vaccine development.


https://twitter.com/intent/like?ref...ism-at-breifing/&tweet_id=1292873241003003904

 

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James Todaro, MD
@JamesTodaroMD
7/ This finding was confirmed in yet another study published in Cell in June 2020 showing that 40-60% of unexposed individuals had T cell recognition of SARS-CoV-2. The authors hypothesized that crossover immunity came from “common cold” coronaviruses.


Targets of T Cell Responses to SARS-CoV-2 Coronavirus in Humans with COVID-19 Disease and Unexposed...
Understanding adaptive immunity to SARS-CoV-2 is important for vaccine development, interpreting coronavirus disease 2019 (COVID-19) pathogenesis, and…
sciencedirect.com
12:19 PM · Aug 10, 2020

Targets of T Cell Responses to SARS-CoV-2 Coronavirus in Humans with COVID-19 Disease and Unexposed Individuals
https://www.sciencedirect.com/science/article/pii/S0092867420306103?via=ihub

Understanding adaptive immunity to SARS-CoV-2 is important for vaccine development, interpreting coronavirus disease 2019 (COVID-19) pathogenesis, and calibration of pandemic control measures. Using HLA class I and II predicted peptide “megapools,” circulating SARS-CoV-2-specific CD8+ and CD4+ T cells were identified in ∼70% and 100% of COVID-19 convalescent patients, respectively. CD4+ T cell responses to spike, the main target of most vaccine efforts, were robust and correlated with the magnitude of the anti-SARS-CoV-2 IgG and IgA titers. The M, spike, and N proteins each accounted for 11%–27% of the total CD4+ response, with additional responses commonly targeting nsp3, nsp4, ORF3a, and ORF8, among others. For CD8+ T cells, spike and M were recognized, with at least eight SARS-CoV-2 ORFs targeted. Importantly, we detected SARS-CoV-2-reactive CD4+ T cells in ∼40%–60% of unexposed individuals, suggesting cross-reactive T cell recognition between circulating “common cold” coronaviruses and SARS-CoV-2.

Graphical Abstract

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2. Download : Download full-size image

 

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False-positive COVID-19 results: hidden problems and costs
https://www.thelancet.com/journals/lanres/article/PIIS2213-2600(20)30453-7/fulltext

 

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False positives in reverse transcription PCR testing for SARS-CoV-2
https://www.medrxiv.org/content/10.1101/2020.04.26.20080911v1.full.pdf

 

[MaddSkillz]

It's a scam and we're allowing it because we're complacent, washed-up, Americans. If HRC was in office, we'd probably hardly even know about it.

 

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Mask Facts
https://aapsonline.org/mask-facts/

Now they recommend wearing cloth face coverings in public settings where other social distancing measures are hard to do (e.g., grocery stores and pharmacies). The recommendation was published without a single scientific paper or other information provided to support that cloth masks actually provide any respiratory protection. Let’s look at the data.

The theory behind mask wearing:
https://aapsonline.org/mask-facts/

this article is packed full of links to actual randomized studies, etc. etc. but will take hours to unpack, never mind reading actual studies linked.

following posts will unpack said data into readable chunks that can be digested in a few minutes.